Search results for " Inbred C3H"

showing 10 items of 55 documents

Circadian and Dopaminergic Regulation of Fatty Acid Oxidation Pathway Genes in Retina and Photoreceptor Cells.

2016

The energy metabolism of the retina might comply with daily changes in energy demand and is impaired in diabetic retinopathy-one of the most common causes of blindness in Europe and the USA. The aim of this study was to investigate putative adaptation of energy metabolism in healthy and diabetic retina. Hence expression analysis of metabolic pathway genes was performed using quantitative polymerase chain reaction, semi-quantitative western blot and immunohistochemistry. Transcriptional profiling of key enzymes of energy metabolism identified transcripts of mitochondrial fatty acid β-oxidation enzymes, i.e. carnitine palmitoyltransferase-1α (Cpt-1α) and medium chain acyl-CoA dehydrogenase (A…

0301 basic medicineMalePhysiologyDopamineMice ObeseGene Expressionlcsh:MedicineBiochemistryAcyl-CoA DehydrogenaseMice0302 clinical medicineCatecholaminesEndocrinologyMedicine and Health SciencesAminesEnzyme Chemistrylcsh:ScienceBeta oxidationMice KnockoutMice Inbred C3HMultidisciplinaryOrganic CompoundsDopaminergicFatty AcidsNeurochemistryDiabetic retinopathyNeurotransmittersCircadian RhythmChemistryCircadian Oscillatorsmedicine.anatomical_structurePhysical SciencesFemaleAnatomyOxidation-Reductionmedicine.drugResearch Articlemedicine.medical_specialtyBiogenic AminesEndocrine DisordersOcular AnatomyBiologyRetinaEnzyme Regulation03 medical and health sciencesOcular SystemInternal medicinemedicineGeneticsDiabetes MellitusAnimalsPhotoreceptor CellsGene RegulationCircadian rhythmCarnitineACADMRetinaDiabetic RetinopathyCarnitine O-PalmitoyltransferaseReceptor Melatonin MT1Receptors Dopamine D4Organic Chemistrylcsh:RChemical CompoundsBiology and Life Sciencesmedicine.diseaseHormonesMice Inbred C57BLMetabolic pathwayDisease Models Animal030104 developmental biologyEndocrinologyMetabolismMicroscopy FluorescenceMetabolic DisordersEnzymologylcsh:Qsense organsEnergy MetabolismPhysiological ProcessesChronobiology030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Efficient and reproducible experimental infections of rats with Blastocystis spp.

2018

Although Blastocystis spp. infect probably more than 1 billion people worldwide, their clinical significance is still controversial and their pathophysiology remains poorly understood. In this study, we describe a protocol for an efficient and reproducible model of chronic infection in rats, laying the groundwork for future work to evaluate the pathogenic potential of this parasite. In our experimental conditions, we were unable to infect rats using vacuolar forms of an axenically cultivated ST4 isolate, but we successfully established chronic infections of 4 week-old rats after oral administration of both ST3 and ST4 purified cysts isolated from human stool samples. The infection protocol …

0301 basic medicinemodèle animal[SDV]Life Sciences [q-bio]lcsh:MedicineBlastocystis Infections[SDV.BC.IC] Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]souris[SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunityFecesblastocyste[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]Medicine and Health SciencesParasite hostingCystratmodèle pour les maladies humaineslcsh:Scienceblastocyst stageProtozoansGastrointestinal tractMice Inbred BALB CMice Inbred C3HMultidisciplinarybiologyaxenic cultureEukaryotaPathophysiologyanimal models3. Good health[SDV] Life Sciences [q-bio]Separation ProcessesExperimental Organism SystemsAnatomyResearch ArticlemiceColonMouse ModelsResearch and Analysis MethodsMicrobiologyculture axeniqueMicrobiology03 medical and health sciencesModel OrganismsmedicineParasitic DiseasesAnimalsHumansClinical significanceAnimal Models of Disease[SDV.IMM.II] Life Sciences [q-bio]/Immunology/Innate immunityDistillationBlastocystisHost (biology)lcsh:ROrganismsBiology and Life Sciencesbiology.organism_classificationmedicine.disease[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyParasitic ProtozoansRatsMice Inbred C57BLGastrointestinal TractChronic infectionDisease Models AnimalAnimal Models of Infection030104 developmental biologyBlastocystisAnimal Studieslcsh:Q[SDV.MP.BAC] Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyParasitic Intestinal DiseasesDigestive System
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Increased expression of the homeostatic chemokines CCL19 and CCL21 in clinical and experimental Rickettsia conorii infection

2013

Background: Based on their essential role in concerting immunological and inflammatory responses we hypothesized that the homeostatic chemokines CCL19 and CCL21 may play a pathogenic role in rickettsiae infection. Methods: Serum levels of CCL19 and CCL21 in patients with R. africae and R. conorii infection were analyzed by enzyme immunoassays. Lungs from R. conorii infected mice were examined for CCL19, CCL21 and CCR7 expression by immunohistochemistry. Results: We found that patients with R. africae infection (n = 15) and in particular those with R. conorii infection (n = 16) had elevated serum levels of CCL19 on admission, with a decline during follow-up. While a similar pattern was seen …

AdultMaleReceptors CCR7Chemokineendocrine systemR. africaeInflammationC-C chemokine receptor type 7ImmunofluorescencePathogenesisMiceYoung AdultmedicineAnimalsHomeostasisHumansAgedInflammationMice Inbred C3HR. conoriiChemokine CCL21biologymedicine.diagnostic_testCCL19Rickettsia InfectionsMiddle Agedbiology.organism_classificationUp-RegulationRickettsia conoriiInfectious DiseasesImmunologybiology.proteinChemokine CCL19FemaleChemokinesmedicine.symptomRickettsia conoriiImmunostainingResearch ArticleCCR7
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Involvement of soluble mediator(s) different from interleukin (IL)1 in the antigen-induced IL 2 receptor expression and proliferation of L3T4+ (CD4+)…

1988

Proliferation of T lymphocytes (T cells) requires the interaction of interleukin 2 (IL 2) with the high affinity form of the IL 2 receptor (IL 2R). IL 2 production as well as IL 2R expression are generally induced simultaneously in T cells by the recognition of specific antigen displayed on the surface of syngeneic antigen-presenting cells. The experiments described herein show that the expression of IL 2R has different requirements than the production of IL 2 (and other lymphokines). Stimulation of antigen-specific L3T4+ T cell lines with antigen-pulsed spleen cells (SC) treated with ultraviolet (UV) light results in efficient IL 2 production but only minimal proliferation due to reduced I…

Antigens Differentiation T-LymphocyteInterleukin 2Ultraviolet RaysT-LymphocytesT cellImmunologyAntigen-Presenting CellsBiologyLymphocyte ActivationMiceInterleukin 21medicineAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigensReceptors ImmunologicInterleukin 3Mice Inbred BALB CMice Inbred C3HLymphokineReceptors Interleukin-2Molecular biologymedicine.anatomical_structureGene Expression RegulationImmunologyInterleukin 12Interleukin-2Cell DivisionSpleenmedicine.drugEuropean Journal of Immunology
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Inducible Co-Stimulator Null MRL-Fas lpr Mice

2005

MRL/MpJ-Tnfrsf6lpr (MRL-Faslpr) mice develop a spontaneous T cell-dependent autoimmune disease that shares features with human lupus, including fatal nephritis, systemic pathology, and autoantibodies (autoAb). The inducible co-stimulator (ICOS) is upregulated on activated T cells and modulates T cell-mediated responses. To investigate whether ICOS has an essential role in regulating autoimmune lupus nephritis and the systemic illness in MRL-Faslpr mice, ICOS null (-/-) MRL Faslpr and ICOS intact (+/+) MRL-Faslpr strains (wild-type [WT]) were generated and compared. It was determined that in ICOS-/- MRL-Faslpr as compared with the WT strain, (1) there is a significant reduction in circulatin…

Antigens Differentiation T-LymphocyteMice Inbred MRL lprT-LymphocytesT cellLupus nephritismedicine.disease_causeBlood Urea NitrogenAutoimmunityInducible T-Cell Co-Stimulator ProteinInterferon-gammaMiceImmune systemimmune system diseasesmedicineAnimalsskin and connective tissue diseasesAutoantibodiesMice Inbred C3HSystemic lupus erythematosusTumor Necrosis Factor-alphabusiness.industryAutoantibodyGeneral Medicinemedicine.diseaseLupus NephritisIsotypeInterleukin-10Mice Inbred C57BLProteinuriamedicine.anatomical_structureNephrologyImmunoglobulin GImmunologyInterleukin-4businessNephritisJournal of the American Society of Nephrology
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Estrogen up-regulates apolipoprotein E (ApoE) gene expression by increasing ApoE mRNA in the translating pool via the estrogen receptor alpha-mediate…

1998

The antiatherogenic property of estrogens is mediated via at least two mechanisms: first by affecting plasma lipoprotein profiles, and second by affecting the components of the vessel wall. Raising plasma apolipoprotein E (apoE) in mice protects them against diet-induced atherosclerosis (Shimano, H., Yamada, N., Katsuki, M., Gotoda, T., Harada, K., Murase, T., Fukuzawa, C., Takaku, F., and Yazaka, Y. (1992) Proc. Natl. Acad. Sci. U. S. A. 89, 1750-1754). It is possible that estrogen may be antiatherogenic at least in part by increasing plasma apoE levels. Therefore, we studied the regulation of apoE by estrogen. A survey of 15 inbred strains of mice showed that some mouse strains responded …

Apolipoprotein EMalemedicine.medical_specialtyApolipoprotein Bmedicine.drug_classEstrogen receptorBiochemistrychemistry.chemical_compoundMiceHigh-density lipoproteinApolipoproteins EInternal medicinemedicineAnimalsRNA MessengerReceptorMolecular BiologyMice Inbred BALB CMice Inbred C3HbiologyEstradiolEstrogen Receptor alphaCell BiologyLipidsUp-RegulationMice Inbred C57BLEndocrinologychemistryGene Expression RegulationLiverReceptors EstrogenEstrogenbiology.proteinlipids (amino acids peptides and proteins)Estrogen receptor alphaLipoproteinThe Journal of biological chemistry
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Activation of phenotypically heterogeneous murine T cell receptor gamma delta + dendritic epidermal T cells by self-antigen(s).

1995

Adult murine epidermis contains a population of Thy-1+, CD45+, CD3+, CD4- and CD8- in situ primarily T cell receptor (TCR) V gamma 3+/V delta 1+ dendritic epidermal T cells (DETC). In the present study, cell surface phenotypes as well as functional properties of DETC were characterized by using in vitro mitogen-stimulated short-term- (10 days) and cloned long-term-cultured (1 year) DETC lines. Phenotypic characterization revealed that80% of the short-term-cultured cells were routinely TCR gamma delta +, CD4-, CD8-. The majority expressed the V gamma 3 TCR. Seventy-five percent of the lines contained detectable numbers of V gamma 2+ (5-7%) and V gamma 2-3- cells (2-3%). Four different types …

CD3T-LymphocytesImmunologyBiologyLymphocyte ActivationCell LineImmunophenotypingInterleukin 21MiceAntigenImmunology and AllergyCytotoxic T cellAnimalsAntigensAntigen-presenting cellMice Inbred BALB CMice Inbred C3HT-cell receptorReceptors Antigen T-Cell gamma-deltaGeneral MedicineDendritic cellT lymphocyteDendritic CellsMice Inbred C57BLImmunologybiology.proteinCytokinesInternational archives of allergy and immunology
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Modulation of proliferation and lymphokine secretion of murine CD4+ T cells and cloned Th1 cells by proteins of the extracellular matrix.

1997

In this study we investigated the co-stimulatory signaling capacity of diverse proteins of the extracellular matrix (ECM) for murine resting CD4+ T cells and Th1 clone cells, activated by immobilized anti-CD3 mAb. ECM proteins used in various concentrations had no effect on IL-2 production or proliferation of highly purified CD4+ T cell populations. When the preparation of CD4+ T cells contained contaminating accessory cells, IL-2 secretion and proliferation was enhanced in the presence of co-immobilized collagens or fibronectin. However, the level of proliferation attainable by added irradiated splenocytes was not reached. Using Th1 cell clone M4, enhanced production of IL-2 in the presenc…

CD4-Positive T-LymphocytesT cellImmunologyLymphocyte ActivationExtracellular matrixInterleukin 21MicemedicineImmunology and AllergyCytotoxic T cellAnimalsSecretionAntigen-presenting cellExtracellular Matrix ProteinsLymphokinesMice Inbred BALB CMice Inbred C3HbiologyChemistryIntegrin beta1LymphokineReceptors Interleukin-2General MedicineTh1 CellsMolecular biologyCell biologyClone CellsFibronectinMice Inbred C57BLmedicine.anatomical_structurebiology.proteinInternational immunology
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Morphological transformation and DNA adduct formation by dibenz[a,h]anthracene and its metabolites in C3H10T1/2CL8 cells.

1994

The major routes of metabolic activation of dibenz[a,h]-anthracene (DBA) have been studied in transformable C3H10T1/2CL8 (C3H10T1/2) mouse embryo fibroblasts in culture. The morphological transforming activities of three potential intermediates formed by metabolism of DBA by C3H10T1/2 cells, trans-3,4-dihydroxy-3,4-dihydro-DBA-(DBA-3,4-diol), trans-dihydroxy-3,4-dihydro-DBA-anti-1,2-oxide (DBA-3,4-diol-1,2-oxide) and DBA-5,6-oxide were determined. DBA-3,4-diol-1,2-oxide was a strong morphological transforming agent giving a mean of 73% dishes with Type II or III foci and 1.63 Type II and III foci per dish at 0.5 microgram/ml. DBA-3,4-diol produced a mean of 42% dishes with Type II or III fo…

Cancer ResearchBiologychemistry.chemical_compoundDNA AdductsMiceStructure-Activity Relationshippolycyclic compoundsmedicineBenz(a)AnthracenesDeoxyguanosineDibenz(ah)anthraceneAnimalsFibroblastCarcinogenBiotransformationMice Inbred C3HGeneral MedicineMetabolismFibroblastsIn vitromedicine.anatomical_structureCell Transformation NeoplasticchemistryBiochemistryCell cultureIsotope LabelingOxidation-ReductionPhosphorus RadioisotopesDNACarcinogenesis
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Coordinate mutation and transformation of mouse fibroblasts: induction by nitroquinoline oxide and modulation by caffeine

1981

Mutation and malignant transformation were followed in the same cells. Mouse fibroblasts (C3H 10T 1/2) were mutated and transformed by 4-nitroquinoline-1-oxide with similar, approximately linear dose-responses. The presence of caffeine immediately after exposure to 4-nitroquinoline-1-oxide potently inhibited mutation and transformation at high but not at low doses of 4-nitroquinoline-1-oxide. Whilst the coordinate induction of mutation and transformation could be explained by both a common target (DNA) or a common reactive species hitting several targets, the identical modulation by a DNA repair inhibitor of both end points suggests fundamental similarities in the nature of the lesions lead…

Cancer ResearchDNA repairDrug ResistanceBiologyMalignant transformationMicechemistry.chemical_compoundCaffeinemedicineAnimalsA-DNAOuabainFibroblastCells CulturedMice Inbred C3HNitroquinolinesDrug SynergismGeneral MedicineMolecular biology4-Nitroquinoline-1-oxideTransformation (genetics)Cell Transformation Neoplasticmedicine.anatomical_structurechemistryMutationMutation (genetic algorithm)Cancer researchCaffeineDNACarcinogenesis
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